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Home - Medicine - A new study found that combining two treatments—one that fixes the faulty SMN2 gene and another that blocks an enzyme called HDAC6—greatly improved muscle strength and survival in a mouse model of spinal muscular atrophy (SMA). This is important because it offers a potential way to help SMA patients who still face muscle weakness even after receiving newer gene therapies.

Medicine

A new study found that combining two treatments—one that fixes the faulty SMN2 gene and another that blocks an enzyme called HDAC6—greatly improved muscle strength and survival in a mouse model of spinal muscular atrophy (SMA). This is important because it offers a potential way to help SMA patients who still face muscle weakness even after receiving newer gene therapies.

Last updated: May 4, 2026 10:20 am
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A new study found that combining two treatments—one that fixes the faulty SMN2 gene and another that blocks an enzyme called HDAC6—greatly improved muscle strength and survival in a mouse model of spinal muscular atrophy (SMA). This is important because it offers a potential way to help SMA patients who still face muscle weakness even after receiving newer gene therapies.
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Researchers discovered that a specific protein called SEL1L3 works as a natural brake on colorectal cancer growth and spread by protecting a key immune signaling molecule called STING from being destroyed inside cells. This finding is significant because it opens up a new target for therapies that could help the body’s own immune system fight colorectal cancer more effectively.
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A powerful new AI system can now generate multiple, high-quality brain MRI images from just a single scan, producing results that doctors considered to be of clinical-grade quality. This breakthrough matters because it could drastically shorten scan times, lower costs, and make advanced brain imaging more accessible and comfortable for patients.
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