How a Single Mutation Can Rewire the Heart’s Molecular Engine
The precise mechanics of cardiac muscle contraction, driven by the myosin motor protein, are fundamental to health and disease. A new biophysical study investigates how genetic mutations that cause cardiomyopathy alter the critical “recovery stroke” of human cardiac β-myosin. This conformational change is essential for positioning the protein’s active site for ATP hydrolysis—the energy source for force generation.
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