Key Highlights
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A new review defines six major hallmarks of cellular senescence, a state where cells stop dividing but remain active, and shows how it can both suppress and promote cancer. This makes targeting senescence a promising new frontier for developing precise cancer therapies.
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Researchers discovered that cancer can be driven by the over-amplification of genes responsible for glycosylation, the process of adding sugar molecules to proteins and fats. This finding expands the list of potential “druggable” targets for cancer treatment to include these sugar-modifying enzymes.
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A study in polycystic ovary syndrome (PCOS) reveals that interconnected cell death pathways are central mechanisms behind impaired egg follicle development. Understanding these pathways provides new potential therapeutic targets for treating this common cause of infertility.
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